Taken from https://sites.duke.edu/harp/our-team/ryan-bell-phd/

Ryan Patrick Bell

Founder & Chair of the Science Policy Committee of the Triangle Chapter of the Society for Neuroscience; and Postdoctoral Associate, Duke University
Areas of Expertise:
  • Health Care
  • Health Care Delivery

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About Ryan

Bell's research interests include investigating the neurobiological effects of HIV infection and co-occurring substance use to identify biomarkers for clinical intervention targets.

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Publications

"Distress Tolerance among Substance Users is Associated with Functional Connectivity between Prefrontal Regions during a Distress Tolerance Task" (with Stacey B. Daughters, Thomas J. Ross, Jennifer Y. Yi, Jonathan Ryan, and Elliot A. Stein). Addiction Biology 22, no. 5 (2017): 1378-1390.

Examines the neural correlates and functional connectivity of distress tolerance among a cohort of substance users and healthy controls. Finds greater activation in a priori cortico-limbic network ROIs, namely the right insula, anterior cingulate cortex, bilateral medial frontal gyrus, right inferior frontal gyrus, and right ventromedial significantly predicted higher distress tolerance among substance users, not healthy controls. 

"White Matter Changes in HIV+ Women with a History of Cocaine Dependence" (with Kathryn-Mary Wakim, Ciara J. Molloy, Lars A. Ross, and John J. Foxe). Frontiers in Neurology 8 (2017): 562.

Uses diffusion tensor imaging to understand the effect of combined HIV+ serostatus and former cocaine dependence on cerebral white matter integrity. Indicates an extensive neuropathological effect of HIV and former cocaine dependence on white matter. 

"Neural Correlates of Craving and Impulsivity in Abstinent Former Cocaine Users: Towards Biomarkers of Relapse Risk" (with Hugh Garavan and John J. Foxe). Neuropharmacology 85 (2014): 461-470.

Examines neural activation patterns in dorsal and ventral striatum in abstinent cocaine dependent individuals and non-using controls as they performed a cocaine craving task. Observes activations in nodes of the response inhibition circuit as they performed an inhibition task.